Claritin-d Drug Uses

Claritin-D is used to relieve hay fever and allergy symptoms, including sneezing, runny nose, and red, itchy, tearing eyes. Claritin-D is in a class of medications called antihistamines. It works by blocking histamine, a substance in the body that causes allergic symptoms. Claritin-D may cause less drowsiness than other antihistamines.

How Taken

Claritin-D comes as a tablet to take by mouth. 12 Hour Tablet: One tablet twice a day (every 12 hours); 24 Hour Tablet: One tablet daily taken with a full glass of water. You should not to break or chew the tablet and you should take the 24 hour tablet with a full glass of water.

Claritin-d Warnings/Precautions

Before taking Claritin-D, tell your doctor if you have asthma or another lung disease; diabetes; thyroid problems; glaucoma or increased pressure in the eye; urinary retention or an enlarged prostate; narrowing of the esophagus or gastrointestinal tract or difficulty swallowing; kidney or liver disease; or heart disease, high blood pressure, or an irregular heartbeat. You may not be able to take Claritin-D, or you may require a dosage adjustment or special monitoring during treatment if you have any of the conditions listed above. Claritin-D is in the FDA pregnancy category B. This means that it is not expected to be harmful to an unborn baby. Do not take Claritin-D without first talking to your doctor if you are pregnant. Claritin-D passes into breast milk and may affect a nursing baby. Do not take Claritin-D without first talking to your doctor if you are breast-feeding a baby. Claritin-D has not been approved for use by children younger than 12 years of age.

Claritin-d Missed Dose

Take the missed dose as soon as you remember it. However, if it is almost time for the next dose, skip the missed dose and continue your regular dosing schedule. Do not take a double dose to make up for a missed one.

Claritin-d Possible Side Effects

Serious side effects are not likely to occur with the use of Claritin-D. If you experience any of the following serious side effects, stop taking Claritin-D and contact your doctor immediately or seek emergency medical attention: an allergic reaction (difficulty breathing; closing of your throat; swelling of your lips, tongue, or face; or hives); an irregular heartbeat; or seizures. Other, less serious side effects may be more likely to occur. Continue to take Claritin-D and talk to your doctor if you experience dizziness; headache; insomnia; nervousness; nausea, diarrhea, or abdominal discomfort; dry mouth; dry skin or itchiness; or discoloration of urine. Side effects other than those listed here may also occur. Talk to your doctor about any side effect that seems unusual or that is especially bothersome.

Claritin-d Storage

Protect Claritin-D from light and store in a dry place. Store between 15-25°C (59-77°F).

Claritin-d Overdose

Seek emergency medical attention. Symptoms of a Claritin-D overdose include headache, drowsiness, an irregular heartbeat, nausea and vomiting, sweating, increased thirst, weakness, and restlessness.

More Information

Use caution when driving, operating machinery, or performing other hazardous activities. Claritin-D may cause dizziness. If you experience dizziness, avoid these

Disclaimer

This drug information is for your information purposes only, it is not intended that this information covers all uses, directions, drug interactions, precautions, or adverse effects of your medication. This is only general information, and should not be relied on for any purpose. It should not be construed as containing specific instructions for any particular patient. We disclaim all responsibility for the accuracy and reliability of this information, and/or any consequences arising from the use of this information, including damage or adverse consequences to persons or property, however such damages or consequences arise. No warranty, either expressed or implied, is made in regards to this information.

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Using Pitrakinra to inhibit interleukins-4 and -13 in the lungs may significantly reduce asthma symptoms, according to an article in the medical journal The Lancet, this week's issue.
The authors explain that doctors and scientists have believed for over twenty years that cytokines, such as interleukin-4 or interleukin-13 play a vital role in the onset and development of clinical asthma. However, there has been no scientific evidence that this might be so.
Dr Malinda Longphre, Aerovance Inc, Berkeley, California, USA and team carried out two separate Phase II clinical trials, where patients received Pitrakinra. In the first study 12 volunteers received 25mg of Pitrakinra by subcutaneous injection once per day. The patients in the second study were given either 60mg of Pitrakinra twice a day by nebulization, and another 16 received a placebo. In both trials the volunteers inhaled allergens before and four weeks after treatment - the allergens are designed to induce an asthma attack, the patient during an asthma attack of this type finds it harder to expel air.
The primary endpoint for the first trial was a maximum percentage drop in forced expiratory volume (FEV1)** in one second over 4-10 hours after receiving the allergen. In the second study the primary endpoint was average percentage fall in FEV1 over 4-10 hours after being given the allergen. If smaller falls in FEV1 during the asthma attack for the Pitrakinra group compared to the placebo group were detected, this would indicate that the allergic response had been reduced by Pitrakinra.
In the first study, say the researchers, the maximum percentage fall in FEV1 was 17.1% in the Pitrakinra and 23.1% in the placebo groups - a comparable difference of 26%. In the second study the average percentage drop in FEV1 was 4.4% in the Pitrakinra versus 15.9% in the placebo group - in other words, it was over three and a half times less in the Pitrakinra group.
"The effects of pitrakinra on late phase asthmatic response are promising when compared with similar studies with other successful anti-inflammatory asthma therapies...whether the effect is due to inhibition of interleukin-13 alone, or both interleukin-13 an interleukin-4, is not yet known. Future studies of this drug, as well as molecules that specifically inhibit interleukin-13, in asthmatic individuals of all levels of severity over longer periods of time are clearly warranted," the writers conclude.

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